This study is part of the collection Swedish Cohort Consortium (Cohorts.se)
Creator/Principal investigator(s)
Margareta Kristenson - Linköping University, Department of Medical and Health Sciences
Description
Also the welfare state of Sweden has prominent socioeconomic (SES) differences in health. These are seen for most measures of SES, i e for education, occupation and income, and also for most health outcomes: for all-cause mortality, for morbidity in most diseases and for self-rated health. SES differences are, in particular, evident for coronary heart disease (CHD) with a two-fold difference in incidence and death between high and low SES groups. Causes for this are not clear. It is well known that an unhealthy lifestyle is more common in low SES but this can only explain a part of observed SES differences.
One possible explanation is effects of psychosocial factors. High levels of psychosocial risk factors and low availability of psychosocial resources are well documented predictors of CHD and more common in low SES. We and others have demonstrated that these factors are related to poor function of the HPA axis with reduced cortisol reactivity and with higher levels of markers for inflammation and plaque vulnerability, which also are known predictors of CHD.
The overall objective of the r
Subject area
HEALTH
(CESSDA Topic Classification)
Medical and Health Sciences
(The Swedish standard of fields of research 2011)
Principal organisation
Accessibility status
Homepage
Responsible department/unit
Linköping University, Department of Medical and Health Sciences
Creator/Principal investigator(s)
Margareta Kristenson - Linköping University, Department of Medical and Health Sciences
Identifiers
SND-ID: EXT 0256
Description
Also the welfare state of Sweden has prominent socioeconomic (SES) differences in health. These are seen for most measures of SES, i e for education, occupation and income, and also for most health outcomes: for all-cause mortality, for morbidity in most diseases and for self-rated health. SES differences are, in particular, evident for coronary heart disease (CHD) with a two-fold difference in incidence and death between high and low SES groups. Causes for this are not clear. It is well known that an unhealthy lifestyle is more common in low SES but this can only explain a part of observed SES differences.
One possible explanation is effects of psychosocial factors. High levels of psychosocial risk factors and low availability of psychosocial resources are well documented predictors of CHD and more common in low SES. We and others have demonstrated that these factors are related to poor function of the HPA axis with reduced cortisol reactivity and with higher levels of markers for inflammation and plaque vulnerability, which also are known predictors of CHD.
The overall objective of the r
Time period(s) investigated
2003-09-30 — 2004-03-03
2012-08-21 — 2015-12-31
Geographic spread
Geographic location: Sweden, Östergötland County, Jönköping County
Unit of analysis
Time Method
Sampling procedure
Ethics Review
Linköping — Ref. 02-324 and 2012/8031
Biobank is connected to the study
Yes
Subject area
HEALTH
(CESSDA Topic Classification)
Medical and Health Sciences
(The Swedish standard of fields of research 2011)
Is part of collection at SND
Associated documentation
Dataset 1
Creator/Principal investigator(s)
Margareta Kristenson - Linköping University, Department of Medical and Health Sciences
Description
Data format / data structure
Numeric
Data collection
Mode of collection: Measurements and tests
Time period(s) for data collection: 2003-09-30 — 2004-03-03
Data collector: Linköping University, Department of Medical and Health Sciences
Source of the data: Population group
Mode of collection: Self-administered questionnaire
Time period(s) for data collection: 2003-09-30 — 2004-03-03
Data collector: Linköping University, Department of Medical and Health Sciences
Source of the data: Population group
Number of individuals/objects
1007
Dataset 2
Creator/Principal investigator(s)
Margareta Kristenson - Linköping University, Department of Medical and Health Sciences
Description
Data format / data structure
Numeric
Data collection
Mode of collection: Self-administered questionnaire
Time period(s) for data collection: 2012-08-21 — 2015-12-31
Data collector: Linköping University, Department of Medical and Health Sciences
Source of the data: Population group
Mode of collection: Measurements and tests
Time period(s) for data collection: 2012-08-21 — 2015-12-31
Data collector: Linköping University, Department of Medical and Health Sciences
Source of the data: Population group
Number of individuals/objects
7051